If you’re experiencing heart attack symptoms, aspirin is the BEST form of first aid. But all aspirins are not alike nor are all methods of taking aspirin alike. Join alongside KRON 4 Morning News anchor, Marty Gonzalez, in taking this lifesaving quiz!
Aspirin can stop an impending heart attack. A heart attack, or myocardial infarction (MI), is permanent damage to the heart muscle. Most heart attacks develop when a cholesterol-laden plaque in a coronary artery ruptures. Plaque deposits are hard on the outside and when this outer shell ruptures (cracks), platelets rush to the area in an effort to ‘patch’ the ruptured area.
Platelets (or “thrombocytes”) are disc-shaped components in the blood that aid in clotting. Aspirin, an anti-platelet, helps inhibit platelet activity. As a clot grows, it blocks an artery. When the artery is completely blocked, cardiac tissue dies from the lack of blood supply and you have a heart attack.
NOTE: Ibuprofen is an anti-inflammatory, and acetaminophen is an analgesic. They are NOT anti-platelet drugs.
A Clot Grows Minute by Minute!
Aspirin can help stop the platelets from forming a larger clot if you take the aspirin BEFORE the clot gets too big. Time is of the essence, so it’s critical to know how the aspirin works the fastest.
QUIZ — Question #1
What is the best DOSE of aspirin to take during a possible heart attack?
Continue reading “KRON 4 | How to Stop a Heart Attack”
Ever wonder what your blood pressure numbers mean? Here’s an explanation using a water faucet and pipe to demonstrate. And now there’s a THIRD pressure that’s important to your overall health and is worth monitoring as well.
What’s Your Pulse Pressure?
Pulse pressure is the numeric difference between systolic and diastolic blood pressure.
Predictor of Heart Attacks and Cardiovascular Disease
A resting pulse pressure in sitting position in healthy adults is about 30-40 mm Hg. For adults over age 60, especially men, a pulse pressure higher than 60 can be a useful predictor of heart attacks or other cardiovascular disease.
- A greater pulse pressure indicates that your vessels are stiffer and more damaged.
- If the aorta becomes rigid, such as from atherosclerosis, a.k.a., “hardening of the arteries”, your pulse pressure would be very high.
- Per a meta-analysis of several studies of 8,000 elderly subjects, the combined results found that a 10-point increase in pulse pressure increased the risk of major cardiovascular complications and mortality by nearly 20%.
NOTE: A low pulse pressure (i.e., 25 mm Hg or less) may mean aortic valve stenosis or congestive heart failure where a low volume of blood is ejected per beat.
Causes of Arterial Stiffness
The most important cause of an elevated pulse pressure is arterial stiffening, in particular, stiffness of the aorta (the largest artery in the body). Arteries stiffen with age, just like tissues in your skin, lungs, heart, tendons, and joints, due to:
- Degeneration of elastin fibers
- Stiffer collagen
- Fatty deposits damaging the arterial walls and resulting in them being less elastic (atherosclerosis)
Results of Arterial Stiffness
- Your heart enlarges. When arteries stiffen and lose their elasticity, your heart must contract more forcefully with each heart beat in order for your blood to circulate adequately. The extra work that your heart must perform can lead to physical changes in the musculature of your heart (e.g., size and shape), specifically the lower chambers (the ventricles). It’s just like how your biceps get bigger that occurs when you exercise them harder.
- You’re prone to irregular heart beats (cardiac arrhythmias). If the remodeling also affects the upper chambers (the atria), it can disrupt the electrical pathways that generate the signals telling your heart when to contract.
- You’re at a higher risk of a heart attack and stroke. The cause of most cardiovascular events and cerebrovascular accidents is the growth and eventual rupture of arterial plaques.
Which Is the Better Pulse Pressure?
160/120 mm Hg OR 110/70 mm Hg? (The pulse pressure in each pair = 40 mm Hg.)
Higher systolic and diastolic pairs imply higher risk of heart attacks and heart disease
Can Atherosclerosis (Coronary Artery Disease) Be Reversed?
Continue reading “KRON 4 | Heart Attack Predictor: A Third Blood Pressure You Need to Know”
VIDEO: You may think you’re one of the lucky ones who can eat unlimited bags of chips and other troves of salt; and yet, it doesn’t affect your blood pressure. But according to research published in the American College of Cardiology, excess sodium can adversely affect other target organs even if you don’t have hypertension.
Studies show elevated sodium levels can cause the inner lining of blood vessels (called the endothelium) to “malfunction”. The following occurs as a result:
- Vessels stiffen. Arteries that become less elastic and lose their contractility (ability to fully contract and relax) make it harder for the heart to pump blood throughout the body. Eventually, the heart wears out (heart failure).
- Vessels become sticky. Think of the lining of your blood vessels as being slick like Teflon. An unhealthy endothelium is sticky like Velcro and causes plaque to attach. Plaque builds up in the brain too — leading to Alzheimer’s.
- The body breeds dangerous inflammation. Inflammation may cause plaque inside blood vessels to crack, rupture, and dislodge from arterial walls. The body responds like with any other injury by causing blood to clot. Clots clog up vessel freeways and block blood flow to the heart (heart attack) or brain (stroke). Inflammation is also the key reason behind why heart attacks are more likely to occur after getting the flu.
NOTE: Eating too much sugar can have the same effect on the endothelium as an excessive intake of sodium.
Research found that elevated sodium levels may increase the wall thickness of the heart’s main pumping chamber (left ventricle). This thickening can lead to cardiac issues, such as:
- Arrhythmia (abnormal heart rhythm)
- Heart attack
- Sudden cardiac arrest (sudden loss of heart function)
- Heart failure (inability of the heart to pump enough blood throughout the body)
High blood pressure is a major risk factor for this muscle tissue enlargement, but it was also found in healthy people with NORMAL blood pressure who consumed high amounts of sodium.
Healthy kidneys remove extra fluid and waste from the blood. Studies found that a high Continue reading “KRON 4 | Salt Affects More Than Blood Pressure”
If you’re experiencing “chest pressure” or “chest pain”, aspirin is the BEST form of first aid. But all aspirins are not alike nor are all methods of taking aspirin alike. Take the lifesaving quiz below…
A heart attack, or myocardial infarction (MI), is permanent damage to the heart muscle. Most heart attacks develop when a cholesterol-laden plaque in a coronary artery ruptures. Plaque deposits are hard on the outside and when this outer shell ruptures (cracks), platelets rush to the area in an effort to ‘patch’ the ruptured area.
Platelets are disc-shaped particles in the blood that aid in clotting. A clot grows minute by minute! As a clot grows, it blocks an artery. When the artery is completely blocked, cardiac tissue dies from the lack of blood supply and you have a heart attack. But aspirin can help stop the platelets from forming a larger clot if you take the aspirin BEFORE the clot gets too big. Time is critical! Aspirin helps inhibit platelet activity.
QUIZ (3 questions):
1. Pick the METHOD that you think makes aspirin work the fastest*. That is, during a suspected heart attack, which of the following is the fastest way to reduce blood clot formation?
A. Swallow the aspirin with 4 oz. of water.
B. Chew the aspirin for 30 seconds, then swallow it.
C. Swallow the aspirin with 4 oz. Alka Seltzer. Continue reading “Heart Attack! First Aid Quiz”
Did you know… that sudden cardiac death is usually the first symptom of coronary heart disease (CHD) — especially among women?
Compared to men, studies show that women are 66% less likely to be diagnosed with coronary heart disease before sudden cardiac death strikes. If you’re a woman and free of symptoms, you’re not identified as “high risk” which means you’re not eligible for cardiac interventions that could save your life. SCD accounts for more than 50% of cardiac deaths (approximately 250,000 to 310,000 cases annually in the United States).
Heart Attack vs. Sudden Cardiac Death
To clarify, the terms “heart attack” and “sudden cardiac death” are NOT the same thing.
- A heart attack or myocardial infarction (MI) occurs when the flow of oxygen-rich blood suddenly gets blocked. Oxygen can’t get to a section of the heart and cardiac tissue dies. Most often the heart is blocked by a build-up of fatty plaque.
- Sudden cardiac death (SCD) is an abrupt loss of heart function as a result of abnormal electrical impulses within the heart. The heart’s electrical system may fail from physical stress, inherited arrhythmias, drug/alcohol abuse, chronic kidney disease, structural changes in the heart, and/or scar tissue that damages the heart’s electrical system. (Cardiac deaths were considered “sudden” if the death or cardiac arrest occurred within 1 hour of the onset of symptoms.)
Simply put, SCD is considered an ‘electrical’ problem whereas a heart attack is more of a ‘plumbing’ problem. Over the years, I’ve had several patients that were revived and survived sudden cardiac arrest who said they didn’t need cardiac rehab because they didn’t have a heart attack, but had an “electrical issue”. They couldn’t be more wrong.
SCD Risk Can Be Prevented
The Journal of the American Medical Association (JAMA) published a 26-year study of nearly 82,000 women that showed you can reduce your risk for sudden cardiac death. In the majority of people, coronary heart disease is usually the underlying cause of SCD and this study showed that a low-risk, healthy lifestyle is associated with a low risk of sudden cardiac death. Continue reading “How to Dodge This Deadly Bullet (Sudden Cardiac Death)”